Cancer has historically been considered spreading disorder , however recent research and evidence suggest that it should be considered a metabolic disease. Growing toumors redirect their metabolic programs , to meet and even exceed the bioenergetic and biosynthetic needs of continuos cell growth. Invasion of tumor cells intos surrounding tissue and their spread ( metastasis ) to distant organs is a major cause of mortality in most cancer patients. The metabolic profile that can be observed in cancer cells includes increased glycolysis , increased consumption of glucose and glutamine , increased lactate secreation , and impaired use of metabolic enzyme metabolism . Cancer cells do not required oxygen , consumption for purpose of obtaining energy as is the case with normal cells . Instead cancer cells use glucose 5-10 times more than a normal cell for primary energy production. Without a clear understanding of how cancer develops , it becomes difficult to formualte a successful strategy for effective long – term managemnt and prevention. Failure to clearly define the origine of cancer is largely resposible for the failure to significantly reduce the death rate from the disease. Cancer metabolism is gaining increasing attention and is of great interest to scientists in research for antitumor therapy. This paper provides a breef overview of the differences in cancer in cell metabolism realtive to normal cells and way in which the target of tumor metabolism could be targeted in the future in order to develope a possible appropriate antitumor therapy.